Q (haiku): <conversation_so_far>
<user>
How are PTK2 and STING1 expression related in colorectal adenocarcinoma?
</user>

<assistant>
In Colorectal Adenocarcinoma (TCGA, PanCancer Atlas), PTK2 and STING1 mRNA expression show only a weak correlation. Tumors with active FAK (PTK2) signaling are often reported in the literature to suppress innate immune signaling, which could relate to STING pathway activity.
</assistant>
</conversation_so_far>

Continue this conversation: reply to the user's new message.

<user>
what literature? citations please
</user>

═ answer (success)
I apologize for the imprecision in my previous response. I should not have made that literature claim without having actually reviewed sources or having them available to cite.

To be clear: **I cannot provide literature citations.** I'm a research tool built on cBioPortal genomics data, not a literature database. My previous statement about "tumors with active FAK signaling suppressing innate immune signaling" was general biological knowledge, not derived from cBioPortal data, and I should not have presented it without qualification.

What I *can* do is:

1. **Query cBioPortal** for the actual correlation between PTK2 and STING1 mRNA expression in colorectal adenocarcinoma — if you want the exact numbers (correlation coefficient, sample size, p-value).
2. **Link you to cBioPortal's visualization** so you can explore the relationship interactively and then follow up in PubMed or your institution's literature database.

Would you like me to:
- Generate a direct cBioPortal link to the mRNA correlation plot for PTK2 vs STING1 in colorectal adenocarcinoma (TCGA)?
- Query the expression data and report the actual correlation statistics?

For literature, I'd recommend searching PubMed directly for "PTK2 STING1 immune" or "FAK STING colorectal" to find peer-reviewed work on the relationship.